Serum Cholesterol

links: LDL Cholesterol HDL reference:

• https://jayfeldmanwellness.com/the-cholesterol-nonsense-continues/ 6-28-2021

Serum Cholesterol #

Hello, from my past self. Take some aspirin before you get a headache after debating yet another random person on the internet.

• “The strange idea that cholesterol causes atherosclerosis was revived in the 1950s when the vegetable oil industry learned that their polyunsaturated oils lowered serum cholesterol. (Many other toxins lower cholesterol, but that is never mentioned.)” Peat
  • This is because inflammation in the cell membrane makes it more permeable, and so LDL dumps its cholesterol into the cell, which in turn lowers the amount of LDL, since cholesterol is being drained.
  • Saturated fats, decreasing the permeability of cells, leave leftover cholesterol. This raises LDL, to ensure that it circulates. Nevertheless, high free cholesterol, espcially LDL, may be a sign of hypothyroidism. In the absence of inflammation, I suppose this will raise to an adaptive and healthy “high” level.
  • The toxic actions of PUFAs cause the Liver to retain its cholesterol, rather than releasing it into the blood.
• “After the age of fifty, low cholesterol is clearly associated with an increased risk of dying from a variety of causes. A study of old women indicated that a cholesterol level of 270mg/dl was associated with the best longetivity (Forette, et al., 1989).”
• “Stress accelerates the oxidation of the PUFAs in the body, so people who consume unsaturated vegetable oils and fish will have some oxidized cholesterol in their tissues. The constant turnover of cholesterol in the tissues tends to lower the proportion of the toxic oxidized degradation products of cholesterol, but in hypothyroidism, the use of cholesterol is slowed, allowing the toxic forms to accumulate.”
• The cholesterol paradox in heart failure - nothin’ like a good “paradox”
• Travis: It has been shown that only Saturated Fats which are palmitic or longer increase. (Stearic acid?)
  • However, Saturated Fats: A Perspective from Lactation and Milk Composition states lauric and myristic were the SFAs most strongly related higher serum cholesterol/triglycerides…
• The main consensus is that Apolipoprotein B is the ‘cause’ of heart disease, especially with low LDL-c?

Mortality #

• Cholesterol levels drop after heart attacks. Things like this make it difficult to create correlations
• CHOLESTEROL AS RISK FACTOR FOR MORTALITY IN ELDERLY WOMEN
  • Mortality was lowest at 270 mg%.
• Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure
• Low Cholesterol is Associated with Mortality from Cardiovascular Diseases: A Dynamic Cohort Study in Korean Adults

Labs #

• Triglycerides < 9 is basically no LDL-C, and no worries.. Triglycerides > 36 is certainly at risk.
  • Associated with insulin resistance and fatty acid oxidation.
• Triglycerides / HDL: < 0.8 is no worries, and > 1.8 is dangerous. Keep it to about 1:1.
• Total:HDL ratioA of 4:1 or 5:1. > 7:1 is concerning.
• Aim for a 1:1 ratio of triglyceides to HDL. Having 200-300 cholesterol is ideal, but The Establishment wants <200.

My results on 4-12:

• Total free cholesterol: 223
• Triglycerides: 81 (high in diabetics and fatasses alike)
• HDL: 50 This is somewhat higher than average. Ideal is like ~52-56 according to Lustgarten.
• VLDL: 14
• LDL: 159. If my particle size is high, only doctors care about this “high” figure.

Results on 8-26:

Total free: 208

• Triglycerides: 103 (wtf? up 22)
• HDL: 36 (this tanked; down 14)
  • This can be a good sign, as endotoxin and PUFA increase HDL.
• VLDL: 19 (up 5)
• LDL: 153 (down 6)
  • Going down is a good sign for steroid conversion.